In Memoriam

Saradindu Dutta, Ph.D. (1931-1999)



For over forty years, Professor Saradindu Dutta sought answers to elusive scientific questions through his academic research. After receiving a Bachelor of Science from the University of Calcutta and a Graduate Degree of Veterinary Science from the Bengal Veterinary College, he emigrated to the United States in 1957. He earned a Master of Science from the University of Wisconsin and Doctor of Philosophy from the Ohio State University.

He spent his career as a professor in the departments of Pharmacology at Ohio State University in Columbus and at Wayne State University in Detroit. Professor Dutta's research in the biochemical aspects of heart and lung functioning led to countless academic publications and collaboration with institutions such as General Motors and the American Heart Association. He supervised the research of doctoral students, he instructed medical student and graduate pharmacology students. His many honors include the Fulbright Scholarship which he received in 1992. He collaborated with international universities, wrote pharmacology review articles and textbook chapters.

As a father, he inspired the pursuit of academic achievement in his children. His son Sumit Dutta, MD and daughter Swati Dutta, MD, received their medical degrees from Michigan State University and the University of Michigan respectively. His son, Suman Dutta, graduated from the University of Michigan in Economics. In his three children, Saradindu Dutta instilled his humor, his kindness and his ethics.

Dr. Dutta was a respected member of the Bengali community of Detroit and was one of their cultural association's founding members. He was a devoted husband for thirty-seven years. He will be missed by his wife Sukla, his children, the Bengali community, his colleagues and former students.

He died in his sleep on third of October, 1999, at six o'clock in the morning. A memorial service was held at the Bharatiya Temple in Troy, Michigan on 24 October 1999.
 

Some selected publications :
 

  • Cullen, WJ, Dulchavsky SA, Devasagayam, TPA, Venkataraman BV, & Dutta, S. Effect of Maharishi AK-4 on H2O2-induced oxidative stress in isolated rat hearts. J. Ethnopharm 56: 215-222, 1997.
  • Dulchavsky SA, Davidson SB, Cullen, WJ, Devasagayam, TPA, Diebel, LN & Dutta, S. Effect of deferoxamine on H2O2-induced oxidative stress in isolated rat heart. Basic Res. Cardiol. 91: 418-424, 1996.
  • Davidson SB, Dulchavsky SA & Diebel LN. Sepsis and T3 replacement improve myocardial integrity during oxidant challenge. Crit. Care Med. 24: 850-854, 1996.
  • Salem YH, Dulchavsky SA, Dutta S & Diebel LN. Effect of inhibition of lipid peroxidation on myocardial oxyradical damage. J. Surgical Res. 56: 606, 1994.
  • Venkataraman BV, Dulchavsky SA & Dutta S. Measurement of physiologic parameters from isolated perfused heart by a computerized system. Indian J. Pharmacology 25:193, 1993.
  • Sen S, Dulchavsky SA & Dutta S. Oxidative stress mediated damage of lungs of ozone exposed rats and its relation to plasma triiodothyronine level. Free Radical Res. Commun. 18: 299, 1993.
  • Dulchavsky SA, Hendrick SR & Dutta S. Pulmonary biophysical effects of triiodothyronine (T3) augmentation during sepsis-induced hypothyroidism. J. Trauma. 35: 104, 1993.
  • Anderson GF & Dutta S. Elctromechanical effects of menadione on isolated rat heart in relation to oxidative stress. Free Radical Biol. Med. 11:169, 1990.
  • Dutta S, Chatterjee M, Teknos TN & Carlson RW. A study of ozone-induced edema in the isolated rat lung in relation to arachidonic acid (AA) metabolism, mixed-function oxidase and angiotensin converting enzyme activities.Pulmonary Pharmacol. 3:65, 1990.
  • Dutta S, Zimmer M, Sies H, Carlson RW, Cullen WJ & Thayer JW. Comparative toxicology of ozone and t-butyl hydroperoxide on isolated rat lung. Free Radical Res. Commun. 8: 27, 1989.
  • Dutta S, Muller A, Ishikawa T, Zimmer M & Sies H. Alkane production by isolated heart and lungs. Toxicology Lett. 44:55, 1988.
  • Chaudhari A & Dutta, A. Effect of an acute exposure of guinea pigs to NO2 on pulmonary prostaglandin dehydrogenase and angiotensin converting enzyme. Bull. Environ. Contam. Toxicol. 25: 816, 1980.
  • Zavecz JH & Dutta S. The relationship between Na+ K+ATPase inhibition and cardiac glycoside-induced cardia arrhythmia in dogs. Naunyn-Schmied. Arch. Pharmac.297: 91, 1977.
  • Dutta S, Marks BH & Schoener EP.Accumulation of radioactive cardiac glycosides by various brain regions in relation to the dysrhythmogenic effect. Brit. J. Pharmac. 59:101, 1977.
  • Stephen PM, Dutta S & Marks BH. The uptake and subcellular distribution of radio-labeled metabolites of digoxin in the isolated perfused guinea pig heart. Naunyn-Schmied. Arch. Pharmac. 292: 251, 1976.
  • Dutta S, Marks BH & Stephen PM. The uptake and subcellular distribution of radio-labeled metabolites of digitoxin in the guinea pig isolated perfused heart.Brit.J.Pharmac. 56:437,1976.
  • Rhee HM, Dutta S & Marks BH.Cardiac Na + K activated ATPase activity during inotropic and toxic actions of ouabain. Europ. J. Pharmac. 37: 141-153, 1976.
  • Dutta, S. and Marks, B.H.: Species and ionic influences on the accumulation of digitalis glycosides by isolated perfused hearts. Brit. J. Pharmacol. 46: 401-408, 1972.

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