Education and Training:

Guangzhou Medical College MD 1980-1985
Guangzhou, China

Medical College of Ohio Ph D 991-1996 Toledo Ohio

Residency:

Surgery Residency 1985-1991
The Second Guangzhou Medical College Hospital
Guangzhou, China

Postdoctoral 1996- 2000
Medical College of Ohio

Pathology Residency (AP/CP) 2000-2004
North Shore Uni-Long Island Jewish Med Ctr Health Syst
New Hyde Park, New York

Fellowship Training:

Clinical Fellow 2004-2005
Renal Pathology
Dept. of Pathology
Johns Hopkins Medical Institute
Baltimore Maryland

Major Interests:

Surgical Pathology, Cytopathology, Tumor Diagnostic Cytopathology and Fine Needle Aspiration

Representative Publications:

1. Zeng, X.; Xie, XH. and Tietz, EI. Impairment of feedforward inhibition in CA1 region of hippocampus after chronic benzodiazepine treatment. Neurosci. lett. 173:40-44, 1994.
2. Zeng, X.; Xie, XH. and Tietz, EI. Reduction of GABA-medicated inhibitory postsynaptic potentials in hippocampus CA1 pyramidal cells following oral flurazepam administration. Neuroscience. 66:87-89, 1995.
3. Zeng, X. and Tietz, EI. Depression of both early and late monosynaptic inhibitory postsynaptic potentials in hippocampus CA1 neurons following chronic benzodiazepine treatment. Synapse. 25:125-136 1996.
4. Zeng, X. and Tietz, EI. Benzodiazepine tolerance at GABAergic synapses on hippocampal CA1 pyramidal cells. Synapse. 31: 263-277, 1999
5. Chen S.; Huang, X.; Zeng, X.; Sieghart, W. and Tietz, EI. Benzodiazepine-mediated regulation of ?1-2, ß1-3 and ?2 GABAA subunit proteins in the rat brain hippocampus and cortex. Neuroscience 93:33-44, 1999.
6. Zeng, X. and Tietz, EI. Role of bicarbonate in mediating conductance in benzodiazepine tolerant hippocampal CA1 pyramidal cells. Brain Research. 868: 202-241, 2000.
7. Tietz, EI.; Zeng, X.; Chen S. Lilly, S.; Rosenberg, H.C. and Kometiani, P Antagonist-induced reversal of functional and structural measures of hippocampal BZ tolerance. J Phamacol Exp. Ther. 291: 932-935 1999.
8. Tietz, EI.; Shanyi C.; Zeng, X. and Sieghart, W. Role of protein kinase A in GABAA receptor dysfunction in CA1 pyramidal cells following chronic benzodiazepine treatment.
   J Neurochem. 85(4):988-98 2003.