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RICHARD
S. VANDER HEIDE, M.D., Ph.D.
Chief
of Pathology and Laboratory Medicine,
VA Medical Center
Education:
Northwestern University Medical School, MD, 1989
Northwestern University Medical School, Ph.D., Experimental Pathology, 1986
Calvin College, B.S., 1981
Training:
Resident, Anatomic Pathology, Duke University Medical Center, Durham, North
Carolina, 1989-1993
Fellow, Cardiovascular Pathology, Duke University Medical Center, Durham, North
Carolina, 1990-1993
Hospital
and Faculty Appointments:
Assistant Professor, Wayne State University, School of Medicine, 1994-2000
Associate Professor, Wayne State University, School of Medicine, 2000-present
Attending Pathologist, Harper Hospital, Detroit, Michigan, 1994-present
Chief of Pathology and Laboratory Medicine, John D. Dingell VAMC, 2000-present
Major Interests:
Ischemic cell injury; cardiovascular pathology; congenital heart disease; autopsy
pathology
Representative
Publications:
1. Vander Heide RS, Ganote CE (1987) Increased myocyte fragility following
anoxic injury. J Mol Cell Cardiol 19: 1085-1103. [Medline]
2. Ganote CE, Vander Heide RS (1987) Cytoskeletal lesions in anoxic myocardial injury: a conventional and high voltage electron microscopic and immunofluorescence study. Am J Pathol 129:327-344. [Medline]
3. Vander Heide RS, Rim D, Hohl C, Ganote CE (1990) An in vitro model of myocardial ischemia utilizing isolated adult rat myocytes. J Mol Cell Cardiol 2: 165-181. [Medline]
4. Vander Heide RS, Reimer KA, Jennings RB (1993). Adenosine slow ischemic metabolism in canine myocardium in vitro : relationship to is chemicpreconditioning. Cardiovasc Res 27:669-673. [Medline]
5. Vander Heide RS, Hill ML, Reimer KA, Jennings RB (1996). Effect of reversible ischemia on the activity of the mitochondrial ATPase: relationship to ischemic preconditioning. J Mol Cell Cardiol 28: 103-112. [Medline]
6. Vander Heide RS, Delyani J, Jennings RB, Reimer KA, Steenbergen C (1996). Reducing lactate accumulation does not attenuate lethal ischemic injury in isolated perfused rat hearts. Am J Physiol 270 (Heart Circ Physiol 39): H38-H44. [Medline]
7. Schwartz LM, Verbinski SO, Vander Heide RS, Reimer KA (1997). Epicardial Temperature is a major predictor of myocardial infarct size in dogs. J Mol Cell Cardiol 29: 1577-1583. [Medline]
8. Hlavaty LA, Vander Heide RS (1998). Causes of isolated aortic insufficiency in an urban population in the 1990's. Cardiovasc Pathol 7:313-319. [Medline]
9. Keller L, Vander Heide RS, Pebbles KA, L'Ecuyer TJ (2000): Percutaneous pulmonary artery gene transfer using naked plasmid DNA delivered via angioplasty. Pediatr Cardiol. 21: 223-227.[Medline]
10. DiCarli M, Precevski P, Singh TP, Leonen M, Muziko O, Vander Heide RS (2000): Myocardial blood flow, function and metabolism in repetitive stunning. J Nucl Med 41(7):1227-1234. [Medline]
11. L'Ecuyer T, Horenstein
MS, Thomas R, Vander Heide RS (2001): Anthracycline-induced cardiac injury
using a cardiac cell line: potential for gene therapy studies.
Mol Genet Metab. 2001 Nov;74(3):370-9. [Medline]
12. Spears JR, Henney C, Prcevski P, Xu R, Li L, Brereton GJ, DiCarli M, Spanta A, Crilly R, Sulaiman AM, Hadeed S, Lavine S, Patterson WR, Creech J, Vander Heide RS (2002): Aqueous oxygen hyperbaric reperfusion in a porcine model of myocardial infarction. J Invasive Cardiol. 2002 Apr;14(4):160-6.[Medline]
13. Vander Heide RS (2002):Increased
expression of HSP27 protects canine myocytes from simulated ischemia-reperfusion
injury. [Medline]
14. Wei H, L'Ecuyer T, Vander Heide RS (2003): Effect of increased expression of cytoskeletal protein vinculin on ischemia-reperfusion injury in ventricular myocytes. Am J Physiol (Heart Circ Physiol) 284: H911-H918.
e-mail: rvanderh@med.wayne.edu